Significados de prevent autoimmunity en inglés

Mesenchymal stem cells have the capacity to maintain immune homeostasis and prevent autoimmunity.

Thus, IL-2R beta is required to keep the activation programs of T cells under control, to maintain homeostasis, and to prevent autoimmunity.

Nevertheless, tumor antigens often are a form of self-antigen, and thus tumor immunity can be dampened by tolerance mechanisms that evolved to prevent autoimmunity.

The rapid and efficient clearance of apoptotic cells results in the elimination of auto-antigens and provides a strong anti-inflammatory and immunosuppressive signal to prevent autoimmunity.

A new study validates the role of these common autoantigens in preventing autoimmunity.

Apoptosis in the immune system is critical for maintaining self-tolerance and preventing autoimmunity.

These interactions may also be important in preventing autoimmunity.

They play critical roles not only in preventing autoimmunity but also in controlling tumor immunity and transplantation tolerance.

Thymic central tolerance is a critical process that prevents autoimmunity but also presents a challenge to the generation of anti-tumor immune responses.

Thus, maintenance of p100 stability by Otub1 serves as an unusual mechanism of NF-kB regulation that prevents autoimmunity.