This may be explained by inflammatory reactions caused by radiation injury.

Probably the two most direct designations are radiation injury and gamma ray injury.

Accordingly, radiation injury is used in this report to mean injury due only to ionizing radiation.

The modification of radiation injury was statistically significant.

Its platform of Toll-like immune receptor activators has applications in mitigation of radiation injury and immuno-oncology.

In contrast, ascorbic acid enhances RelB expression in normal cells, improving antioxidant and metabolic defenses against radiation injury.

We investigated the ability of docetaxel to enhance in vivo tumor radioresponse and influence radiation injury to normal tissue.

Patients experience reduced neurological morbidity from GKS compared with open microneurosurgery, but risks of radiation injury and technical limitations persist.

The average annual incidence of radiation injury at FMLH was 13.8%.

The average annual incidence of radiation injury among gynecologic cancer patients at ASLH was 5.5%.

Interleukin 1 (IL-1) has been shown to protect a number of normal tissues against radiation injury.

When tested in vivo, we show that human MEMs significantly enhance survival from lethal GVHD and improve survival of mice from radiation injury.

In mouse jejunum, modest protection against radiation injury has been reported using only the in vivo crypt survival assay in one mouse strain.

The aim of this review is threefold: First, to make a short brief into the natural history of radiation injury to the intestinal tract.

To date, no drugs have been approved as radiation injury mitigators, which are defined as agents administered after irradiation but before toxicity is manifest.

The aim of this study was to investigate the influence of overall treatment time on the development and repair of radiation injury in the bladder.