The process in which a myeloid precursor cell acquires specializes features of an erythrocyte.
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These results indicate that constant production of CO from heme regulates erythropoiesis.
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Antihypertensive therapeutics and factors influencing erythropoiesis are negatively associated with HTS formation.
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Defective terminal erythropoiesis contributes to decreased generation of RBCs in many disorders.
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We review the molecular roles of BETs in the context of erythropoiesis.
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Several mechanisms by which iron regulates erythropoiesis were also recently identified.
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One mode of heme-regulated transcription involves amplifying the activity of GATA-1, a key determinant of erythrocyte differentiation.