Background: Calcification of renalallografts is common in the first year after transplantation and is related to hyperparathyroidism.
9
Results: In this period 146 children received 183 renalallografts.
10
The subgroup analysis by RIC type demonstrated no significant difference among the three interventions in protecting renalallografts against DGF.
11
Background: Increased oxygen radical production may not only contribute to posttransplant ischemia-reperfusion injury but also to acute rejection of renalallografts.
12
These results support the continued approach of providing both cadaver-donor and living-donor renalallografts for recipients whose primary renal allograft has failed.
13
Alloreactive IFN-gamma-producing PBLs derive from the memory T cell pool and are readily detectable in recipients of renalallografts taking immunosuppressant medications.
14
Eculizumab appears to be effective in protecting renalallografts when post-transplant aHUS or AMR occur, although the published cases report relatively short follow-up.
15
Patients with failing renalallografts often progress to end-stage renal disease, unlike transplant-naive chronic kidney disease patients, in an accelerated and unpredictable manner.
16
These results indicate that the mechanism of CTL killing leading to the rejection of renalallografts may be different in acute and chronic rejection.