We assessed the effect of CAI in dextran sodiumsulfate-induced colitis.
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The disruption of the epithelial barrier with dextran sodiumsulfate leads to increased IL-19 expression.
3
Separation was optimized by altering pH, the concentration of sodiumsulfate and the pairing ion.
4
Recent findings: Using the azoxymethane-dextran sodiumsulfate model, wound healing pathways seem to be required in the development of CAC.
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Functional studies showed that defective IL-36R signalling causes high susceptibility to acute dextran sodiumsulfate colitis and impairs wound healing.
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A third common colitis model, administration of dextran sodiumsulfate, was hopelessly confounded by the high sodium content of the dextran sodiumsulfate.
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The results showed that EFC can be used to convert 95% of the wastewater stream to pure ice (98% purity) and sodiumsulfate.
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Studies in genetically engineered mice showed that epithelial STAT3 activation in dextran sodiumsulfate colitis is dependent on interleukin (IL)-22 rather than IL-6.