Significados de volatile anesthetics en inglés

It is well known that both local and volatile anesthetics depress myocardial functions.

A mutation in gas-1 profoundly increases sensitivity of C. elegans to volatile anesthetics.

However, they may contribute to the increased sensitivity to volatile anesthetics of the gas-1 mutant.

Oxidative damage to proteins may be an important contributor to sensitivity to volatile anesthetics in C. elegans.

We demonstrated in the past that volatile anesthetics such as sevoflurane attenuate ischemia-reperfusion injury during liver resection.

Currently, mouse models describing the impact of volatile anesthetics on cancer growth are limited to same-sex models.

So far, it is unknown whether postconditioning elicited by volatile anesthetics can also provide protection in the remodeled myocardium.

The effect of volatile anesthetics on cancer cell survival and death after anesthetic exposure has not been well investigated.

Also, cardiac or pulmonary complications, increased rhabdomyolysis risk when exposed to volatile anesthetics and succinylcholine may increase anesthesia related risks.

A mutation in the gene gas-1 alters sensitivity to volatile anesthetics, fecundity, and life span in the nematode Caenorhabditis elegans.

The impact of volatile anesthetics on cancer progression has been observed for decades, but sex differences have not been described.

In this randomized study, we examined if volatile anesthetics have an effect on acute graft injury and clinical outcomes after liver transplantation.

Previous studies have shown that antagonism of glutamate at the NMDA receptor reduces the minimum alveolar concentration (MAC) for volatile anesthetics.

Preconditioning with hyperbaric oxygen, volatile anesthetics, and xenon seems to be the focus of the attention from the standpoint of the clinical setting.

Conclusion: These findings indicate that the use of halogenated volatile anesthetics modulates the type I IFN response to influenza and enhance postinfection antibacterial immunity.

In addition, volatile anesthetics have been shown to accelerate postischemic neurogenesis; this suggests that anesthetics may enhance the endogenous reparative processes in the injured brain.