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Thyroid hormones are permissive for various species to enter seasonal anestrus.
2
In addition, reproductive hormone levels were determined during ovine anestrus and the breeding season.
3
Thyroid hormones permit the increase in response to estradiol negative feedback in ewes at the transition to anestrus.
4
Furthermore, the levels of estrogen, progesterone and LH in ovine anestrus were significantly lower than those in the breeding season.
5
This finding contrasts with the changes in estradiol negative feedback at the transition to anestrus, which are entirely thyroid hormone dependent.
6
Thus, the premammillary region and vmPOA are brain sites in which thyroid hormones act to permit the onset of seasonal anestrus.
7
Both doses delayed puberty; low E also produced pronounced prepubertal and seasonal anestrus hypogonadotropism, and delayed the onset of the second breeding season.
8
In the ewe they act centrally to permit the onset of potent estradiol-negative feedback responsible for anestrus, but the specific sites of action are unknown.
9
Therefore, we have proposed that estrogen-responsive afferents to A15 neurons are part of the neural circuit mediating E2 negative feedback in anestrus.
10
The IUD caused anestrus in 98% of the heifers, with a daily weight gain 25.5 % higher than in the control heifers.