Background: Increased aortic stiffness contributes to systolic hypertension and increased cardiovascular risk.

Conclusions: Increased AI is not a reliable surrogate for increased aortic stiffness.

Carotid-femoral pulse wave velocity was used as a measure of aortic stiffness.

Overall, worsening metabolic traits were associated with worsening aortic stiffness and MAP.

However, the prevalence and correlates of abnormally high aortic stiffness are incompletely understood.

There were good correlations between LV remodelling and aortic stiffness values.

We investigated the effect of caffeine on aortic stiffness in treated hypertensive patients.

Models evaluating aortic stiffness also were adjusted for mean arterial pressure.

Conclusion: Dysglycaemia independently predicted aortic stiffness after five years in older black adults.

A number of clinical studies have analyzed the effects of age on aortic stiffness.

We reported earlier that excessive alcohol use predicts aortic stiffness.

Metabolic syndrome (MS) is related to increased aortic stiffness in several populations.

Conclusions: Caffeine affects unfavorably aortic stiffness and enhances wave reflections.

Carotid-femoral PWV was determined in order to assess aortic stiffness.

However, the exact mechanism leading to elevated aortic stiffness in patients with COPD is unknown.

Conclusions: Asymptomatic WHIV on ART have increased aortic stiffness as compared to matched control subjects.