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1 Background: The inflammatory response plays a critical role in hypertension-induced cardiac remodeling .
2 Background: Myocardial infarction leads to cardiac remodeling and development of heart failure.
3 Background: Tobacco smoke exposure is an important risk factor for cardiac remodeling .
4 These findings may shed light onto obesity-related cardiac remodeling and heart failure.
5 The molecular events underlying these distinct forms of cardiac remodeling are poorly understood.
6 However, its role in cardiac remodeling after myocardial infarction has not been elucidated.
7 Prior studies showed that wall stress is a vital parameter of cardiac remodeling .
8 Oxidative stress is widely involved in pathophysiological processes of cardiac remodeling .
9 Molecules associated with antioxidant functions may be ideal targets for reversing cardiac remodeling .
10 We investigated decorin's chronic effects on postinfarction cardiac remodeling and dysfunction.
11 Fibrosis, a prominent feature of pathological cardiac remodeling , manifested partial reversibility.
12 Objective: Inflammatory responses play a pivotal role in the pathogenesis of hypertensive cardiac remodeling .
13 Studies showed the significant relationship between OSA and cardiac remodeling .
14 This evidence of cardiac remodeling was present in obese children as young as age 8.
15 This review discusses some recent advances in understanding the role of autophagy during cardiac remodeling .
16 These data demonstrate that MLK3 prevents adverse cardiac remodeling in the setting of pressure overload.
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This collocation consists of: Cardiac remodeling across language varieties