Significados de cellular redox em inglês

Hypoxia and cellular redox state are among the key factors that drive wound angiogenesis.

The detoxification of ROS also is an important function of the cellular redox homeostasis system.

Genetically encoded redox probes enable the functional analysis of individual proteins in cellular redox homeostasis.

The induction of HO-1 restores cellular redox processes, which contributes to inhibition of the toxic milieu.

Furthermore, it provides additional evidence suggesting that torsinA is sensitive to changes in the cellular redox potential.

NRF2, a transcription factor that has been deemed the master regulator of cellular redox homeostasis, declines with age.

We also predict that complex I helps maintain a cellular redox state by reoxidizing NADH produced from central metabolism.

This observation emphasizes the importance of subcellular compartmentalization in understanding the biology of the cellular redox system in plants.

These findings suggest that the activation of lysosomal function protects against anlotinib-mediated cell apoptosis via regulating the cellular redox status.

However, when cellular redox balance is severely disturbed, high levels of ROS may cause various damages leading to cell death.

While under normal conditions most cellular compartments maintain a reducing environment, the cellular redox state can be influenced by external factors.

We conclude that the cellular redox state may be involved in cellular transformation caused by antioxidants such as melatonin and trolox.

NAD plays critical roles in cellular redox reactions and remains at a sufficient level in the cell to prevent cell death.

Its regulation includes control of reactive oxygen species (ROS) generation, sensing deviation from and readjustment of the cellular redox state.

We show that PFKFB4 is required to balance glycolytic activity and antioxidant production to maintain cellular redox balance in prostate cancer cells.

The studies demonstrate that cellular redox changes can directly regulate gene expression in vivo by determining the level of occupancy of strategically positioned GC-binding sites.