CT was performed because of a posterior cerebralinfarct caused by cardiac arrhythmia.
2
We suggest that SCS is a predictor of symptomatic cerebralinfarct in patients with AF.
3
The case-fatality rate for CH is three to four times that for cerebralinfarct (CI).
4
Neurological deficit scores, cerebralinfarct volume, and morphological characteristic were detected at corresponding time after cerebral ischemia.
5
Rats showed behavioral deficits as assessed with neuroscore that correlated with cerebralinfarct size and retinal function at 2days.
6
As a result, T2-weighted images demonstrated neither cerebralinfarct nor intracerebral hemorrhage, but identified abnormal dilatation of the lateral ventricles in SHR-SP.
7
Methods: A retrospective review was performed for patients undergoing DHC for cerebralinfarct from 2016 to 2019.
8
The exception was that only men showed a positive relationship between executive function and cerebrovascular disease defined as large WMH volume plus silent cerebralinfarct.
9
Diffusion-weighted magnetic resonance imaging (DWI-MR) revealed a focal high intensity signal in the right precentral gyrus at high convexity with a cerebralinfarct.
10
Increased expression of MMP-9 was associated with degree of stenosis and presence of cerebralinfarct on CT scan (p=0.05).
11
Cerebralinfarct volume was similar in the valproic acid and baicalin groups.
12
No association between dietary protein intake and silent cerebralinfarcts was found.
13
MRI of the brain demonstrated multiple cerebralinfarcts through they were asymptomatic.
14
No association of major dietary protein sources with silent cerebralinfarcts was detected.
15
MetS was not associated with an increased occurrence of WMH or cerebralinfarcts.
16
Rapid MRI of the molecular diffusion of water demonstrated cerebralinfarcts in 32 patients.