1
A restriction site was present for analysis of the codon 41 mutation.
2
Bacillus subtilis possesses three classes of genes, differing by their codon preference.
3
Ribosome profiling provides the opportunity to evaluate translation kinetics at codon level resolution.
4
Also, codon context patterns vary among these species largely according to their phylogeny.
5
However, the stop codon is preceded by a run of 11 consecutive adenosines.
6
For example: the codon AAC makes asparagine, the codon GCA makes alanine, etc.
7
The single-base pair mutation causes a frameshift, which results in premature stop codon.
8
The triplet codon repeats lead to heterochromatin-mediated gene silencing and loss of frataxin.
9
This duplication results in a frame shift and premature stop codon.
10
A premature stop codon UGA appeared to be most responsive to PTC124 treatment.
11
The alternative in-frame start codon is not present around a mutation.
12
K-ras codon 12 mutations were detected using PCR followed by allele-specific oligonucleotide hybridization.
13
Surprisingly, there is a strong negative correlation between codon usage and protein length.
14
RNA structural prediction showed the codon optimization removed two potential RNA pseudoknot structures.
15
Protooncogene Ha-ras codon 12 mutations are frequently observed in thyroid cancers.
16
PDR prevalence was calculated by demographics and codon, stratifying by prior ARV experience.
