Both can result from perturbations in the flux of iron across the absorptive intestinal enterocyte.
2
Small bowel mucosal structural changes and enterocyte epithelial apoptosis were determined 48 h following laparotomy.
3
This study was designed to examine the effect of metformin on glucose transporters in enterocyte.
4
However, the enterocyte warns the host of impending danger and, in turn, elicits a protective response.
5
Moreover, we predict that the presence of the small intestinal enterocyte induces competition over host-derived nutrients.
6
To verify the library, clones containing a complete locus of enterocyte effacement (LEE) were identified by DNA hybridization.
7
In contrast, eel enterocyte plasma membrane and renal brush border membranes contain only the pI 6.5 form.
8
These results indicate that COX-2 is an important chemoprevention target and that inhibition of this enzyme alters a paracrine enterocyte regulatory pathway.
9
Furthermore, h-OVA induced lower activities of serum mast cell protease-1 and enterocyte brush border membrane alkaline phosphatase as compared to native OVA.
10
In conclusion, metformin slightly increases intestinal glucose absorption by inducing a re-distribution of glucose transporters in BBM through AMPK control in enterocyte.
11
Intestinal amino acid profiles and 13C tracer appearance into these pools were significantly altered, indicating abnormal amino acid trafficking through the enterocyte.
12
Thus, in a game of constant attack and defence, the pathogen and the enterocyte aim to outsmart each other in an effort to survive.
13
Rejection was confirmed by histological study of the explanted intestine, enterocyte apoptosis was determined in crypts and the lamina propria of the small bowel.
14
Moreover, the binding affinities of ezetimibe and several key analogs to recombinant NPC1L1 are virtually identical to those observed for native enterocyte membranes.
15
We have examined the effects of leptin on PepT1 function in rat jejunum and in monolayers of the human enterocyte-like 2 cell Caco-2.
16
These results indicate that RIP1 plays a major role in physiological enterocyte turnover through a RIP3-independent nonapoptotic death mechanism in the mouse small intestine.