Accumulation of too much fat inside liver cells.
Sinônimos
Examples for "hepatosteatosis"
Examples for "hepatosteatosis"
1In our study, we detected mild hepatosteatosis in all hypothyroidism-induced rats.
2However, the composition of liver LDPs and the role of their alterations in hepatosteatosis are not well-understood.
3In conclusion, we found that hypothyroidism-induced rats had mild hepatosteatosis as opposed to the control group histopathologically.
4Here, we summarize recent literature reporting dissociation of hepatosteatosis from insulin resistance in genetic mouse models and clinical studies.
5A reduction in liver fibrosis and hepatosteatosis was evident in RB394-treated obese ZSF1 rats.
1However, it may prevent worsening of hepatic steatosis associated with weight gain.
2The fatty-acid saturation of the diet hardly affected hepatic steatosis or inflammation.
3HCV-associated IR may also cause hepatic steatosis and resistance to antiviral treatment.
4Moreover, exercise can effectively reduce hepatic steatosis independent of changes in body mass.
5Studies are needed to determine whether treatment of hepatic steatosis reduces systemic inflammation.
1Metabolic changes due to nonalcoholic fatty liver disease may explain this relationship.
2An autopsy showed he suffered from a fatty liver and enlarged heart.
3The mechanism determining the progression from fatty liver to steatohepatitis is unknown.
4This observation suggests a novel mechanism for the pathogenesis of fatty liver.
5The presence and severity of fatty liver was assessed by abdominal ultrasonogram.
6Conclusions: We modeled the natural history of fatty liver for adult Japanese men.
7Resolution of NAFLD was defined as absence of ultrasound criteria for fatty liver.
8Background: Obesity-related fatty liver disease is linked to mitochondrial dysfunction and oxidative stress.
9Methods: Mice were fed the Lieber-Decarli ethanol diet to induce alcoholic fatty liver.
10Previously, we demonstrated that development of fatty liver depends on adipocyte GH signaling.
11This model induced a fatty liver with focal necrosis and fibrosis.
12Muc2 deficiency protected mice from HFD-induced fatty liver disease and obesity.
13The pathophysiologic continuum of non-alcoholic fatty liver disease begins with steatosis.
14The highest rate of fatty liver was seen in obese children (38%).
15However, it is unclear whether leptin improves fatty liver independently of these metabolic improvements.
16Abdominal obesity and dyslipidemia are the important risk factors for nonalcoholic fatty liver disease.