Elimination of Na+ or HCO3- also caused marked decreases in uptake.
2
However, how human pancreatic duct cells secrete copious amounts of HCO3- has long been a puzzle.
3
Ongoing bone loss was linearly related to arterialized HCO3, even after adjustment for age and renal function.
4
A supply of protons from the nonbicarbonate buffer pool is necessary to maintain a high rate of HCO3- dehydration.
5
Carbonic anhydrases II and IV play an essential role in the synthesis and secretion of HCO3- ions in pancreatic duct cells.
6
The GABA-induced increase in aiCl which took place in the presence of HCO3- was blocked by clamping the membrane potential at its resting level.
7
Results: Serum HCO3 increased in all patients and final values did not differ statistically between the two groups so results for all patients were combined.
8
Using the defined medium we have additionally shown that shedding is related to HCO3- concentration under conditions of controlled medium pH, Cl- content and osmolality.
9
Inhibition of astroglial HCO3- transport may be a critical and requisite event, ultimately leading to compartmentalized astroglial acidosis and irreversible injury to all cell types.
10
With respect to groundwater chemistry, the fluoride concentration was highest in Na-HCO3 type groundwater and lowest in Ca-HCO3 type groundwater.