Significados de induce nephropathy em inglês

Conclusion: This study suggests that BG protects or ameliorates against contrast- induced nephropathy.

This increase did not seem to be caused by cyclosporine- induced nephropathy.

Caspase-12-dependent apoptotic pathway may be partially involved in the urografin- induced nephropathy.

This toxic effect subjects in the disorder of CM- induced nephropathy.

However, radiation dose and the risk of contrast induced nephropathy are major downsides of CTA.

The gradual excretion process could prevent contrast- induced nephropathy.

There is evidence that calcium antagonists may have a beneficial effect on cyclosporine- induced nephropathy after transplantation.

Background: Contrast- induced nephropathy (CIN) is associated with poor outcomes in patients with acute myocardial infarction.

Contrast- induced nephropathy (CIN) is a common clinical problem for which there is no effective medical treatment.

Background: Contrast- induced nephropathy (CIN) refers to acute renal damage that occurs after the use of contrast agents.

Background: Contrast- induced nephropathy (CIN) is a serious condition in patients with ST-segment-elevation myocardial infarction treated with primary percutaneous coronary intervention.

We advocate using contrast-enhanced computed tomography as the primary diagnostic imaging modality as concern about intravenous contrast agent- induced nephropathy is relatively minor.

This study investigated the renal protective effect of probucol in a rat model of contrast- induced nephropathy and the mechanism of its effect.

History: A 64-year-old obese man with radiocontrast- induced nephropathy, which required temporary hemodialysis, was referred for treatment of an infrarenal abdominal aortic aneurysm detected incidentally.

Taken together, these findings suggested that the acute phase of AA-I- induced nephropathy is associated with induction of Atg5-dependent autophagy, which promotes renal tubular cell apoptosis.

Similarly, we did not observe significant differences between wild-type or IL-22-deficient mice in a mouse model of secondary focal and segmental glomerulosclerosis (adriamycin- induced nephropathy).