1However, their functions during the influenza A virus infection process remain unclear.
2Background: During March 2009 a novel Influenza A virus emerged in Mexico.
3Avian influenza A viruses often do not propagate efficiently in mammalian cells.
4Thirty-four chinchillas were inoculated intranasally with either influenza A virus or sterile saline.
5Pandemic influenza A virus (IAV) remains a significant threat to global health.
6Water birds are the natural host of influenza A viruses.
7Our data indicate that NA alone among viral proteins limits influenza A virus superinfection.
8Sulfonic acid polymers exert selective inhibitory effects on RSV and influenza A virus replication.
9In summary, this study establishes a quantitative biochemical correlate for influenza A virus transmission.
10However, to what extent sumoylation is exploited by influenza A virus is not fully understood.
11Taken together, these findings indicate that autophagy is actively involved in influenza A virus replication.
12However, little is known about the regulatory role of autophagy in influenza A virus replication.
13Rapid antigenic evolution in the influenza A virus hemagglutinin precludes effective vaccination with existing vaccines.
14Novel antivirals are needed to supplement existing control strategies for influenza A virus (IAV).
15It was recently reported that influenza A virus proteins interact extensively with the host sumoylation system.
16By comparison, the influenza A virus has been found to survive on surfaces for 17 days.
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Translations for influenza a virus