Similar differences in mitochondrial morphology were observed in glucose-limited chemostat cultures.

We also investigated changes in mitochondrial morphology, which can directly influence mitochondrial function.

GDAP1-specific knockdown by RNA interference results in a tubular mitochondrial morphology.

Indeed, alterations in mitochondrial morphology, distribution and function are common features in catabolic conditions.

However, whether the mitochondrial morphology in myeloid immune cells affects anti-tumor immunity is unclear.

Conditional and inducible Opa1 deletion alters mitochondrial morphology and function but not DNA content.

Furthermore, mitochondrial morphology from cardiac and skeletal muscle was altered.

Background and aims: Recent reports have described dramatic alterations in mitochondrial morphology during metazoan apoptosis.

Emerging evidence suggests that oxidative stress disturbs mitochondrial morphology dynamics, resulting in detrimental mitochondrial fragmentation.

We also show that the mitochondrial morphology affects Parkin expression and its recruitment to mitochondria.

Loss of Pdx1 leads to abnormal mitochondrial morphology and function as well as impaired mitochondrial turnover.

Significant alteration in mitochondrial morphology was also detected.

The mitochondrial morphology was observed by electron microscopy.

Recently, altered mitochondrial morphology and a deficit in the energetic activity in FA cells have been reported.

These results identify PERK-dependent SIMH as a protective stress-responsive mechanism that regulates mitochondrial morphology during ER stress.

Similarly, there were no differences in mitochondrial morphology or myocardial fibrosis between mdivi-1 and vehicle control groups.