Significados de nonalcoholic fatty em inglês

Metabolic changes due to nonalcoholic fatty liver disease may explain this relationship.

Abdominal obesity and dyslipidemia are the important risk factors for nonalcoholic fatty liver disease.

Conclusion: Metabolic syndrome might significantly affect the degree of nonalcoholic fatty liver in children.

In nonalcoholic fatty liver disease the amount of fibrosis for individual histologic stages is unknown.

Mechanisms underlying progression of nonalcoholic fatty liver disease (NAFLD) are still incompletely characterized.

Iron is suspected to enhance hepatic damage associated with nonalcoholic fatty liver disease (NAFLD).

Inflammation and oxidative stress are considered critical factors in the progression of nonalcoholic fatty liver disease.

The beneficial effect of CWE was evaluated in a murine model of nonalcoholic fatty liver disease.

A condition defined as nonalcoholic fatty liver disease (NAFLD) is frequently found in humans.

The leading reason for why potential living liver donors are not eligible is nonalcoholic fatty liver disease.

Liver fibrosis is the most important predictor of mortality in nonalcoholic fatty liver disease (NAFLD).

Modulation of adipokines may aid in further understanding of the pathophysiology and treatment of nonalcoholic fatty liver disease.

Liver biopsy is currently the only reliable method to establish nonalcoholic fatty liver disease (NAFLD) severity.

The fibrotic potential of nonalcoholic fatty liver disease is as severe as that of chronic hepatitis C virus.

It is speculated that thyroid hormones may be involved in nonalcoholic fatty liver disease (NAFLD) pathogenesis.

Although nonalcoholic fatty liver has a negligible risk of progression, patients with NASH often develop cirrhosis or hepatocellular carcinoma.