1
Heart failure was induced in rabbits by combined volume and pressure overload.
2
However, the cardiac function worsened upon ageing and when subjected to pressure overload.
3
Conversely, IRF4 knockout mice showed an attenuated hypertrophic response to chronic pressure overload.
4
Malonyldialdehyde production induced by pressure overload was suppressed by quercetin.
5
Then, we examined the responses of both ventricles to chronic pressure overload in vivo.
6
Its underlying pathophysiology differs from that of cardiac pressure overload.
7
Conclusions: MAFbx plays an essential role in mediating cardiac hypertrophy in response to pressure overload.
8
Methods and results: Cardiac hypertrophy was induced by slow progressive pressure overload in adult cats.
9
Chronic systemic hypertension causes cardiac pressure overload leading to increased myocardial O(2) consumption.
10
These data demonstrate that MLK3 prevents adverse cardiac remodeling in the setting of pressure overload.
11
We aimed to investigate the impact of RDN on cardiac angiogenesis during prolonged pressure overload.
12
Finally, we examined cardiac responses to chronic pressure overload by transverse aortic constriction in vivo.
13
In this study, cardiac dysfunction and hypertrophic remodeling in mice were induced by pressure overload.
14
Hypertrophy was induced in young cats by slow progressive pressure overload of the feline right ventricle.
15
A model of chronic, progressive pressure overload of the left ventricle was characterized in the cat.
16
Herein, we investigate the role of MKK7 in regulating cardiac remodeling in response to pressure overload.
