1
Autophagy and apoptosis play significant roles in PRRSV infection and replication.
2
Our previous research suggested that HO-1 may play an important role in PRRSV infection.
3
And inhibition of the complement system likely attenuated the lung damage during HP- PRRSV infection.
4
However, the function of HO-1 in PRRSV infection is unclear.
5
PRRSV infection was determined and virus strains were classified by the sequencing of GP5.
6
In addition, PRRSV infection induced a significant increase of microRNA editing level in both breeds.
7
We subsequently demonstrated that the NF-kB signaling pathway was essential for PRRSV infection-induced IL-15 production.
8
Our findings might provide new insights into the molecular mechanisms underlying the pathogenesis of HP- PRRSV infection.
9
Our findings indicate that delivery of miR-181 can be used as antiviral therapy against PRRSV infection.
10
These results presented here provide a basis for understanding molecular mechanism of PRRSV infection and inflammation response.
11
Previous research showed that only a few BMo became susceptible to PRRSV infection after 1 day culture.
12
Our findings provide new insights into the molecular mechanisms underling the IL-15 production induced by PRRSV infection.
13
It was observed that miRNAs and genes show different expression patterns at different time points during PRRSV infection.
14
Our data obtained here could provide crucial clues for better understanding the roles of NSP12 in PRRSV infection.
15
We then found that NF-kB was indeed activated upon PRRSV infection, as evidenced by IkB phosphorylation and degradation.
16
Northwest China has a sparse pig population and there is no comprehensive information currently available on PRRSV infection.