Significados de septic patients em inglês

This information has been considered in the clinical management of septic patients.

Results: Differences in glucose uptake and storage were significantly less in septic patients.

Methods: Twenty-five consecutive septic patients were divided randomly into two groups.

The relationship between AUD and cortisol in septic patients is unknown.

Adipose tissue biopsies and circulating leukocytes were collected from septic patients.

Higher cortisol increases risk of death in septic patients.

Conclusion: G-CSF causes leukocyte stiffness but attenuates inflammatory response without inducing lung injury in septic patients.

Despite the many advances in the treatment of sepsis, mortality rates in septic patients remain high.

Patients: Cells from healthy donors and septic patients.

In addition to sepsis, pharmacological agents used to treat septic patients may contribute to mitochondrial dysfunction.

Treatment of septic patients with anti-mediator strategies or high dose AT III were not successful so far.

Temporal trends and the distribution of pathogens in identified septic patients during the aforementioned period were studied descriptively.

Conclusion: G-CSF administration was effective in septic patients with a low percentage of immature neutrophils and insufficient endogenous G-CSF.

Findings of impaired oxygen utilization in septic patients and animals implicate nitric oxide-mediated inhibition of the mitochondrial respiratory chain.

The levels of circulating miR-15 family members are elevated in septic patients and may be associated with septic death.

This was in contrast with TNFa which decreased production was illustrative of the endotoxin tolerance phenomenon occurring in septic patients.