Propranolol and atenolol had no significant effect on the serumglucose levels.
2
Results: MIT treatment significantly attenuated fat accumulation, serumglucose levels, and excessive cholesterol.
3
Significant elevations were observed in serumglucose but not insulin levels by maltose infusion.
4
Overexpression of STAMP2 ameliorated IR and decreased serumglucose level.
5
However, few data compare the simultaneous effects of elevated serumglucose and aging on AD.
6
Blood samples were collected to analyze serumglucose, cholesterol, triglycerides, urea, and total protein levels.
7
Compared to unexposed controls, serumglucose levels rose during exposure and returned to control levels postexposure.
8
His serumglucose was extremely high at 853.
9
A significant elevation of serumglucose-AGE was found to be associated with severity of diabetic retinopathy.
10
Rapamycin elevated serumglucose in female diabetic mice.
11
One group of diabetic animals was treated with slow release insulin to maintain normal serumglucose levels.
12
In contrast to HOMA-IR and fasting serum insulin, no significant changes in fasting serumglucose were observed.
13
Therefore, we tested the hypothesis that serumglucose, insulin, cholesterol, and triglyceride levels would correlate with gallbladder contractility.
14
High serumglucose concentrations are known to induce the polyol pathway and increase fructose generation in the liver.
15
O3 increased serumglucose and leptin on day 1.
16
In addition, we have previously demonstrated that serumglucose, insulin, cholesterol, and triglycerides correlated with gallbladder contractility in murine models.