Cholera is an acute intestinal infection caused by the Vibriocholerae bacillus.
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Here we investigate defense against biofilm invasion using the model bacterium Vibriocholerae.
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Vibriocholerae is a Gram-negative bacillus that is the causative agent of cholera.
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Cholera is caused by a water-borne bacteria called Vibriocholerae.
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Vibriocholerae uses a variety of strategies for obtaining iron in its diverse environments.
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The human pathogen and aquatic bacterium Vibriocholerae belongs to the group of naturally competent bacteria.
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Vibriocholerae is a human pathogen, which is transmitted by the consumption of contaminated food or water.
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However, confusion still surrounds the relationships between globally circulating pandemic Vibriocholerae clones and local bacterial populations.
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In this study we report the effects of iron and Fur on gene expression in Vibriocholerae.
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Vibriocholerae, the causative agent of the severe diarrheal disease cholera, thrives in both marine environments and the human host.
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We test the predictions of our model using the enteric pathogen Vibriocholerae, which produces an extracellular matrix important for biofilm formation.
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Here we report the crystal structure of the N-terminal cytoplasmic domain of EpsF, an inner membrane spanning T2SS protein from Vibriocholerae.
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Our data pave the way for potential use of ϕJA1 and ϕVchO139-Iin Vibriocholerae typing and control.
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Cholera outbreaks South Africa has faced several of these bacterial infection outbreaks transmitted through the consumption of water polluted with the Vibriocholerae bacteria.
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Importance: Vibriocholerae is a major human pathogen and also serves as a model for the Vibrionaceae, which include other serious human and fish pathogens.
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The transcription factor Fur regulates the expression of a number of genes in Vibriocholerae in response to changes in the level of available iron.