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Up to one-third of the COA patients we studied are overweight.
2
In primary cortical neuron cultures, COA-Cl prevented neuronal injury after 2h of oxygen-glucose deprivation.
3
Delayed continuous COA-Cl administration also reduced infarct volume.
4
Only the COA group had higher characteristic impedance.
5
The more active COA patients report higher scores of perceived physical functioning, general health, and mental health.
6
Conclusions: Adults with repaired COA have a reduced exercise tolerance, which is related to low physical activity levels.
7
The results thus suggest the major contribution of S1P1 in the angiogenic effects of COA-Cl.
8
The effect of COA-Cl was evaluated in vivo with 60min of middle cerebral artery occlusion combined with bilateral common carotid artery occlusion.
9
Importantly, a COA focused on opioid craving could be a valuable addition to research studies designed to evaluate novel treatments for OUD.
10
We performed in vitro and in vivo experiments to investigate whether COA-Cl can attenuate neuronal damage and enhance recovery after brain ischemia.
11
Background: Patients with aortic coarctation (COA) have a reduced exercise capacity and seem to be more prone to adopt a sedentary lifestyle.
12
Changes in the cell morphology consistent with EMT were induced by TGF-β1, whereas, this response was suppressed by co-treatment of the cells with COA-Cl.
13
Our results demonstrated that the treatment of cells with COA-Cl suppressed the TGF-β1 mediated increase in the mRNA levels of ZEB2.
14
Cell-culture studies using inhibitors of HMG-CoA reductase have suggested an immunosuppressive effect.
15
The significance of other acetyl-CoA sources, however, has not been rigorously evaluated.
16
COAs were given Diagnostic Interview Schedule alcohol diagnoses more frequently than non-COAs.