Post hoc the relationship with lifetime alcohol use disorder was explored.

No association was found between cognitive functioning and lifetime comorbid alcohol use disorder.

Ultimately, this provides mechanistic insight into the etiology and pathophysiology of alcohol use disorder.

The greatest effects were associated with alcohol use disorder.

There was no statistically significant association between PPU with suicidal ideation or alcohol use disorder.

Patients with severe depressive symptoms, (hypo)manic symptoms and current severe alcohol use disorder were excluded.

Excessive serotonin (5-HT) signaling plays a critical role in the etiology of alcohol use disorder.

However, men and those with alcohol use disorder might benefit from interventions targeting pain interference to reduce polysubstance use.

The alcohol use disorder identification test (AUDIT) and self-reported ethanol intake in the last 28 days were assessed.

Recent studies have indicated that patients with prior bariatric surgery (BS) are at risk for developing alcohol use disorder.

Moderating effects of family history density of alcohol use disorder (FHD) on these associations and sex differences were explored.

Purpose: To determine the efficacy of behavioral and pharmacological interventions for insomnia among individuals with alcohol use disorder (AUD).

Mostly heterosexual women also had higher levels of past-year alcohol use disorder symptomology, recent tobacco and marijuana use, and depressive symptoms.

More severe subgroups showed significantly higher level of anxiety, depression, hostility, obsessive-compulsivity, interpersonal sensitivity, and psychiatric or alcohol use disorder-related treatment involvement.

Previous research suggests that the DSM-IV alcohol use disorder (AUD) symptoms in adolescents may be characterized by a single dimension.

In all alcohol use disorder patients with ALD psycho-social therapy and attendance at SHG groups it is mandatory, even in post-transplant period.