Male sex hormone that is produced in the testes and responsible for typical male sexual characteristics.
1 Furthermore, treatment with NA causes reduction of androgen - mediated AR target gene expression.
2 Conclusions: Collectively, the results establish RKIP as a novel androgen target gene.
3 In case of adverse situations androgen deprivation therapy, chemotherapy, hormonal therapy, etc.
4 After combined androgen blockade treatment, median CRPC occurrence time was 23 months.
5 No significant changes were observed in Ferriman Gallwey score or androgen levels.
6 Mutations in the AR gene prevent the androgen receptors from working properly.
7 The lesional cells strongly expressed cytoplasmic cytokeratin 7 and nuclear androgen receptor.
8 In a few patients, aberrant regulation of androgen excess has been reported.
9 Several randomized trials of androgen supplementation in older men have been undertaken.
10 The expression of the androgen receptor was also suppressed by the treatment.
11 Remarkably, Q tract variation also differentially impacts disease progression following androgen depletion.
12 The associations appeared to be modified by circulating estrogen and androgen levels.
13 Surprisingly, androgen receptor controls a splicing program distinct from its transcriptional regulation.
14 The compound also decreased the expression of the androgen receptor and PSA.
15 A subset of these androgen - regulated proteins appeared to be expressed in abundance.
16 Conclusions: PC-SPES is a well-tolerated and active treatment for androgen - independent prostate cancer.
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Translations for androgen
Androgen в диалектах
Соединенные Штаты Америки