1
Intervention that suppressed this signaling loop may potentially retard age-associated adverse arterial remodeling.
2
Pulmonary arterial remodeling and constriction are two prominent features of PAH.
3
Objective: Coronary atherosclerosis has been associated with systemic arterial remodeling even in nonatherosclerotic vessels.
4
However, there are few data that correlate plaque morphology, composition, and arterial remodeling in vivo.
5
The mechanisms of lumen enlargement during stent implantation may be significantly affected by arterial remodeling.
6
Black blood imaging was performed to quantify coronary wall thickness and indices of arterial remodeling.
7
Changes in matrix gene expression and persistence of treatment effects after drug withdrawal suggest arterial remodeling.
8
This review mainly focuses upon the landscape of MFG-E8 expression and signaling in adverse arterial remodeling.
9
Background: Chronic changes in blood flow stimulate arterial remodeling, which contributes to the maintenance of vascular homeostasis.
10
Thus, interventions to restrict proinflammatory signaling are a rational approach to delay or prevent age-associated adverse arterial remodeling.
11
However, little is known about the plaque features and arterial remodeling in the increased atherogenesis caused by SR-BI deficiency.
12
Age-associated arterial remodeling involves arterial wall collagen deposition and elastin fragmentation, as well as an increase in arterial pressure.
13
These results confirm that directionally guiding SMC responses after injury achieves favorable arterial remodeling and limits development of pathologic neointima.
14
At the end of treatments, the hemodynamic parameters and pathological right ventricular and pulmonary arterial remodeling were analyzed in each group.
15
Conversely, attenuating Ang II signaling via an inhibition of angiotensin conversing enzyme or a blockade of AT1 activation retards age-associated arterial remodeling.
16
Most intravascular ultrasound (IVUS) studies of arterial remodeling in native coronary arteries reported a remodeling index obtained at a single time point.
