1 However, the stimuli to AVP is likely different between cycling and running.
2 Results: Twenty-two patients were found to carry a pathogenic AVP - NPII gene mutation.
3 One AVP patient developed acute coronary syndrome with dose-dependent ECG changes.
4 No differences were found for non-social working memory or for AVP vs placebo.
5 We assessed the results of vascular embolization obtained using the AVP .
6 The magnitude of the AVP - induced vasoconstriction was significantly reduced by calphostin-C.
7 The modest decrease in PV was not the primary non-osmotic stimulus to AVP .
8 The contents of SS and AVP in hippocampus tissue were measured by radioimmunoassay.
9 Conclusions: Peripheral embolization with the AVP 4 was successful in the majority of patients.
10 At the maximum effect, pinacidil completely relaxed vasoconstriction in the continuing exposure to AVP .
11 These results suggest that periventricular dopaminergic mechanisms may act to inhibit hemorrhage-induced AVP secretion.
12 The copeptin assay may be a useful alternative to direct measurement of AVP concentration.
13 This study evaluated the AVP 4 in peripheral vascular embolization.
14 Measurement of AVP levels has limitations due to its short half-life and cumbersome detection method.
15 In ICU patients, AVP decreased to moderately elevated levels within 24 h after ED admission.
16 It has been proposed that this is because AVP facilitates baroreflex control of the circulation.
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