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The huskies faced the direction of the sounds, moving restlessly and whining.
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The huskies who were gathered about the room immediately chorused their disapproval.
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Then followed a detoxified Conservative Party, hugging hoodies and frolicking with huskies.
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And many thanks to Joan Irwin, for rescuing me from the huskies.
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Unlike David Cameron, who used to want to hug hoodies and save huskies.
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The different uterine drug accumulation of VP16 and L-V contributed to partly cause for the functional variation.
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We generated transgenic mice expressing a constitutively active VP16-LXRa protein from the aP2 promoter.
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Moreover, VP16-LXRa expression also suppressed the induction of inflammatory genes by lipopolysaccharide to a comparable degree as synthetic agonist.
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The herpesvirus VP16 transactivator has become a useful tool for facilitating the production of recombinant proteins in cultured mammalian cells.
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We demonstrate that 24 h of exposure to XK469 before VP16 resulted in a maximum synergistic response.
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Three distinct forms of VP3 were identified among the nine human rotavirus strains analyzed.
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In conclusion, the VP3-ELISA is a sensitive and specific method for detecting antibodies against GPV or MDPV.
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A 66-year-old man was diagnosed with hepatocellular carcinoma with Vp3 by abdominal enhanced CT.
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Another mutation in the VP3 protein resulted in altered immunological properties of the isolates, possibly facilitating virus spread in immunized populations.
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Genetic characterization of the viruses revealed that amino acid 60 of the VP3 region was mutated in all AD P1 isolates.
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The neoplasm subsequently metastasized and required additional VP-16 and carboplatin for control.
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Cisplatin and VP-16 combination can be considered as a salvage treatment in heavily pretreated patients with advanced breast cancer.
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Fas-FasL system participated in the apoptosis induced by DEX and VP-16; different drugs induce apoptosis by different pathway of signal transduction.
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Chromatin condensation was morphologically evident only when cells detached from the monolayer; untreated or VP-16-treated attached cells retained a normal morphology.
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This effect was even more pronounced when cells were exposed to VP-16 4 hours before gemcitabine.
1
There were differences in the molecular weights of viral proteins (VP) 3 and 4 from the two serotypes, and serotype II isolates lacked VP-2.
Translations for bombing squadron one hundred thirty six