Значения термина brain infarction на английском

Conclusion: Coronary atherosclerosis and age were important risk factors for silent brain infarction.

The frequency of silent brain infarction increased with the severity of coronary stenosis.

Subclinical silent cerebrovascular disease was defined as silent brain infarction and white matter hyperintensity volume.

The present results suggest that aFGF may be involved in the repair processes following brain infarction.

Objective: Silent brain infarction is fairly common in the elderly, but predictive factors have not been definitively established.

He had chronic constipation and had taken aspirin for about 10 years because of a previous brain infarction.

This study focuses attention on ischemic heart disease and cerebrovascular risk factors about the frequency of silent brain infarction.

In a predominantly older population-based cohort, both brachial and central pulse pressure were independently associated with silent brain infarction.

Conclusions: Diffusion-mapping imaging may help to examine brain infarction from 3 hours to 5 days.

From January 2007 to June 2008, 323 consecutive patients with acute brain infarction were prospectively studied.

Postoperative brain infarction in cervical fusion could be a complication of the usually safe lateral mass plating of the cervical spine.

The results of the present study demonstrated that YC-1 effectively reduced brain infarction and brain edema, and improved blood-brain barrier leakage.

Elevation of plasma fibrinogen combined with a loss of high-density lipoprotein cholesterol is strongly associated with severe atherosclerosis and results in brain infarction.

However, prolonged ischemia resulted in greater brain infarction and increased neurological deficits in addition to greater reductions in the ipsilateral and the contralateral SSEPs.

Consistent with this finding, selective (PPARγ) genetic deletion in vascular endothelial cells resulted in increased cerebrovascular permeability and brain infarction in mice after focal ischaemia.

Brain infarction and hemorrhage within infarcts were measured, and neurological deficits were scored.