Значения термина cardiac progenitor cell на английском

These proteins are indicated as potential targets to control cardiac progenitor cell fate by materials design.

Here we demonstrate that the paralog proteins YAP and TAZ exert a crucial role in adult cardiac progenitor cell mechano-sensing and fate decision.

However, previously described cardiac progenitor cell populations show immature development and need co-culture with neonatal rat cardiomyocytes in order to differentiate in vitro.

C-kit positive cardiac progenitor cells increased their numbers in exercise-induced physiological hypertrophy.

C-kit and Sca-1 positive cardiac progenitor cells were activated by swimming training.

Conclusion: This study presents that swimming-induced physiological hypertrophy initiates activation of cardiac progenitor cells.

In pregnancy, c-Kit positive cardiac progenitor cells were activated.

Immunofluorescent staining was used to compare the number of C-kit and Sca-1 positive cardiac progenitor cells.

Background: Endogenous cardiac progenitor cells are a promising option for cell-therapy for myocardial infarction (MI).

Collectively, these results permit enhancement of stepwise differentiation and facilitate isolation and expansion of cardiac progenitor cells.

Background: In recent years, resident cardiac progenitor cells have been identified in, and isolated from the rodent heart.

Our study provides evidence that receptor-kinase-dependent EphB4-forward signaling plays a crucial role in the development of cardiac progenitor cells.

Conclusion: This study presents that pregnancy-induced physiological hypertrophy activates cardiac progenitor cells and thereafter protects against cardiac ischemia-reperfusion injury.

Rationale: Myocardial function is enhanced by adoptive transfer of human cardiac progenitor cells (hCPCs) into a pathologically challenged heart.

Studies on cardiac progenitor cells (CPCs) and their derived exosomes therapeutic potential have demonstrated only modest improvements in cardiac function.

Sca-1 positive cardiac progenitor cells were increased in LV and OFT in mice swimming for 3 weeks.