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However, the roles ubiquitous factors play in the specification of cellidentity remain underappreciated.
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In combination with molecular instructions, the physical principles behind cellidentity determination are examined.
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Therefore, stabilizing β- cellidentity upon isolation may improve its functionality.
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Gene regulation by transcription factors (TFs) determines developmental programs and cellidentity.
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Most cellidentity dissection studies are based on bulk molecular assays that mask differences in individual cells.
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Cellular products derived from the activity of DNA, RNA, and protein synthesis collectively control cellidentity and function.
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Contrary to predictions, we find that Dppa4 is completely dispensable for ES cellidentity and germ cell development.
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Conclusions: We have identified the first molecular components that control lateral root founder cellidentity in the Arabidopsis root.
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Many studies have addressed how local signals from neighboring niche cells regulate stem cellidentity and their proliferative potential.
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These differences are required to establish and maintain differences in cellidentity, cell function, tissue homeostasis, and tumor suppression.
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These findings highlight the importance of chromatin mapping in understanding unique features of stem cellidentity and stem cell aging.
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The histological and scanning electron microscope analyses revealed cellidentity changes in both external and internal tissues of Alq sepals.
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Moreover, ions released from these mineral-based biomaterials play a vital role in defining cellidentity, as well as driving tissue-specific functions.
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Th17 cellidentity is determined by a spectrum of extracellular signals, including cytokines, which are critical orchestrators of cellular immune responses.
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We identified two distinct and synergistic transcriptional modules that dominate successful reprogramming, which are associated with cellidentity and biosynthetic genes.