1
Fast nicotinic transmission might play a greater role in cholinergic signaling than previously assumed.
2
Conclusions: Remodeling of cholinergic signaling is a critical pathological component of human and canine HF.
3
Acetylcholine is locally produced in the thymus and cholinergic signaling influences the T cell development.
4
In contrast, cholinergic signaling in the septohippocampal system is suggested to be involved in memory processes.
5
Although β-adrenergic signaling has been studied extensively, the effect of CRT on cholinergic signaling is unexplored.
6
The relative contribution to brain cholinergic signaling by synaptic- and diffusion-based mechanisms remains to be elucidated.
7
Therefore, drugs that target AChE are likely to be important regulators of cholinergic signaling in the hippocampus.
8
However, the relationship between cholinergic signaling during cue detection and neural activity dynamics in prefrontal networks remains unclear.
9
The purpose of this study was to determine if interruption of autocrine muscarinic cholinergic signaling has potential to inhibit SCLC growth.
10
Differential remodeling of cholinergic signaling represents a novel mechanism for enhancing sympathovagal balance with CRT and may identify new targets for treatment of systolic HF.
11
Here we show that selenium-induced oxidative stress leads to decreased cholinergic signaling and degeneration of cholinergic neurons required for movement and egg-laying in Caenorhabditis elegans.
