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Resting muscle concentrations of ATP and phosphocreatine did not change.
2
During cardioplegia, myocardial ATP and phosphocreatine contents increased and glycogen concentration tended to decrease.
3
Significant correlations were observed between endomyocardial and transmural adenine nucleotide and glycogen contents but not phosphocreatine content.
4
Creatine, a nitrogenous organic acid, replenishes cytoplasmic ATP at the expense of mitochondrial ATP via the phosphocreatine shuttle.
5
CKMT1 inhibition altered mitochondrial respiration and ATP production, an effect that was abrogated by phosphocreatine-mediated reactivation of the arginine-creatine pathway.