Значения термина epithelial to mesenchymal transition на английском

Recent studies have associated epithelial to mesenchymal transition with the development of chemoresistance.

Additionally, there was significant upregulation of genes associated with epithelial to mesenchymal transition.

FAM3C is a secreted factor, which is involved in the epithelial to mesenchymal transition.

Increased Src activity promotes this process and inhibition of Src suppresses epithelial to mesenchymal transition.

Proteins that regulate cell proliferation, invasion, and epithelial to mesenchymal transition are among the identified ADAM12-interacting proteins.

Understanding the epithelial to mesenchymal transition may explain the aggressive pattern of spread frequently observed in this disease.

This review focuses on upregulation of Src in cancer as it relates to chemoresistance and epithelial to mesenchymal transition.

Recent evidence has demonstrated that epithelial to mesenchymal transition (EMT) is associated with chemoresistance in pancreatic carcinoma cells.

We find no evidence at the cellular or molecular level for epithelial to mesenchymal transition of labeled cells into myofibroblasts.

The epithelial to mesenchymal transition (EMT) is a developmental process enabling epithelial cells to gain a migratory mesenchymal phenotype.

Although multiple Src functions may contribute to metastasis, recently Src has been shown to play a role in epithelial to mesenchymal transition.

These cells appear to separate from the anterior epithelium and undergo a dramatic change that is reminiscent of the epithelial to mesenchymal transition (EMT).

Background: The microRNA-200 family participates in the maintenance of an epithelial phenotype and loss of its expression can result in epithelial to mesenchymal transition (EMT).

The epithelial to mesenchymal transition (EMT) plays crucial roles in the formation of the body plan and in the differentiation of multiple tissues and organs.

We further demonstrated that depletion of HEIRCC reduce the epithelial to mesenchymal transition (EMT) program by regulating expression levels of EMT-associated markers in RCC cells.

Genome-wide analysis of H3K79me2 distribution revealed that fibroblast-specific genes associated with the epithelial to mesenchymal transition lose H3K79me2 in the initial phases of reprogramming.