Значения термина follow tbi на английском

Conclusion: Evidence for WM impairments following TBI is consistent with previous research.

This idea may provide new avenues of clinical assessments and treatments following TBI.

Lastly, this review addresses the potential contribution of other proteases to neuronal damage following TBI.

From these studies we conclude that following TBI Pioglitazone is capable ameliorating multiple aspects of neuropathology.

We hypothesized that the presence of TWMA defined by MTI would correlate with outcome following TBI.

Programmed death of neuronal cells plays a crucial role in acute and chronic neurodegeneration following TBI.

The sequalae that follow TBI result in neurological dysfunction across a host of physiological and psychological domains.

The results provide a novel herbal medicine to protect BBB following TBI via an antioxidative molecular mechanism.

Studies investigating the neurochemical basis for the protracted cognitive dysfunction that follows TBI have focused in part on cholinergic mechanisms.

The results revealed that the optimal dose of NIM811 improves cognition, improves mitochondrial functioning, and reduces oxidative damage following TBI.

This article reviews new evidence indicating that overactivation of calpains plays a major role in the neurodegenerative cascade following TBI in vivo.

The high calcium permeability of the alpha7 nAChr may be related to the extensive decrease in BTX binding that occurs following TBI.

Therefore, a novel small-molecule tool was used to target CRMP2 in an attempt to determine its importance in mossy fiber sprouting following TBI.

These results suggested that TGFβ1 played a protective role in axonal injury and could be a potential therapeutic target in early stages following TBI.

This review provides an update on the quantitative mutimodal neuroimaging studies that attempt to elucidate the mechanism(s) underlying cognitive impairments and their recovery following TBI.