Mutations in voltage- gated ion channels are responsible for several types of epilepsy.

Correolide displays marked selectivity against numerous receptors and voltage- and ligand- gated ion channels.

Voltage- gated ion channels are critical to excitation-secretion coupling in nerve terminals.

Animal and plant voltage- gated ion channels share a common architecture.

These ligand- gated ion channels are crucial in the control of cell and network activity.

Ionotropic neurotransmitter receptors and voltage- gated ion channels assemble from several homologous and non-homologous subunits.

Ligand- gated ion channels play a role in mediating fast synaptic transmission for communication between neurons.

Voltage sensors have been well studied in voltage- gated ion channels for neuronal excitation and muscle contraction.

Pardaxins physically interact with cell membranes by forming pores or voltage- gated ion channels that disrupt cellular functions.

Most CRISPs can inhibit ion channels, such as the cyclic nucleotide- gated ion channel, potassium channel, and calcium channel.

This environment provides ligands that have recently been shown to act as pharmacological chaperones in the biogenesis of ligand- gated ion channels.

There is growing evidence indicating that the pore structure of voltage- gated ion channels (VGICs) influences gating besides their conductance.

The local neural dynamics is determined by voltage- and ligand- gated ion channels and feedback between densely interconnected excitatory and inhibitory neurons.

Neuronal and cardiac voltage- gated ion channels contain auxiliary subunits that can profoundly affect the gating of the pore-forming and voltage-sensing alpha subunits.

After the discovery of Channelrhodopsin, a light- gated ion channel, only a few people saw the diverse range of applications for such a protein.

The roles of cells within the nervous system are based on their properties of excitability, which are in part governed by voltage- gated ion channels.