Мы используем Cookies Этот веб-сайт использует cookie-файлы, чтобы предлагать вам наиболее актуальную информацию. Просматривая этот веб-сайт, Вы принимаете cookie-файлы.
However, the heart retains the ability to return to the "fetal" geneprogram.
2
Despite the advance, there is no therapy available to reverse the already committed geneprogram.
3
In contrast, exposure to arterial laminar flow induces a geneprogram that confers a largely antiadhesive, antithrombotic effect.
4
Finally, real-time quantitative PCR revealed an age-dependent recapitulation of the fetal geneprogram, consistent with a myopathic process.
5
Finally, we show that OXT acts directly on OXTRs on adipocytes to suppress the white-to-beige transition geneprogram.
6
NF-kappaB regulates the fetal geneprogram and PPARalpha-regulated gene expression during cardiac hypertrophy and induces atrophy in skeletal muscle.
7
The absence of calsarcin-1 also activated a hypertrophic geneprogram, despite the absence of hypertrophy, and enhanced the cardiac growth response to pressure overload.
8
In response to stress signals, postnatal cardiomyocytes undergo hypertrophic growth accompanied by activation of a fetal geneprogram, assembly of sarcomeres, and cellular enlargement.
9
We propose that in failing heart muscle at a certain point the fetal geneprogram is no longer sufficient to support cardiac structure and function.
10
The HDAC3-PROX1 module controls the expression of a geneprogram regulating lipid homeostasis, and hepatic-specific ablation of either component increases triglyceride content in liver.
11
Nrf2 is the key transcription factor for cytoprotective geneprograms.
12
The liver coordinates the expression of geneprograms that control glucose absorption, storage, and secretion.
13
Unstable angina may be a clinical analogue to ischemic preconditioning, and involve activation of geneprograms.
14
In this review, we will discuss how TCR-signaling events coordinate cellular metabolism and specialization geneprograms in T cells.
15
Transcription factors are essential nuclear proteins that trigger the expression of geneprograms required for long-term functional and structural plasticity changes.
16
These findings provide further support for an important role of LXRs in the coordinated regulation of lipid metabolic and inflammatory geneprograms in macrophages.