Exposure of tissues to abnormally high concentrations of oxygen.
1 MnTBAP reduced vessel density and failed to improve alveolar growth following hyperoxia .
2 Retinal vascular density measurements revealed no signs of neovascularization after hyperoxia exposure.
3 Cell death caused by exposure to hyperoxia was decreased in HK-II-overexpressing cells.
4 These studies demonstrate that early activation of Akt occurs in hyperoxia in HLMVEC.
5 Lung injury was not observed when the exposure to hyperoxia preceded bleomycin treatment.
6 It also provided reproducible responses to systemic hypoxia and hyperoxia over this time period.
7 Ventilatory and MSNA responsiveness to hyperoxia and hypoxia were not significantly correlated within individuals.
8 In the mouse, neonatal hyperoxia decreased the number of c-KIT+ EC progenitors.
9 Neonatal hyperoxia impairs vascular and alveolar growth in mice and decreases endothelial progenitor cells.
10 Subacute hyperoxia may cause basement membrane disruption and subsequent fibrosis.
11 We have shown that hyperoxia reduces brain damage in a rat model of hypoxia-ischemia.
12 The method reproducibly detected acute changes in cortical oxygenation induced by systemic hyperoxia and hypoxia.
13 Results: Angiogenic gene expression was significantly lower in the hyperoxia group compared to the RA group.
14 The 4-d exposure to hyperoxia or retinoids did not lead to observable differences in lung development.
15 The purpose of this study was to examine the possibility of hyperoxia in inducing vision-threatening retinopathy.
16 Special attention is paid to the mechanisms by which hyperoxia affects these transcription factors in the lung.
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Translations for hyperoxia
Hyperoxia в диалектах
Соединенные Штаты Америки