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Fourteen cases of congenital hyperparathyroidism secondary to maternal hypocalcemia have been reported.
2
IP is significantly associated with temporary hypocalcemia, but not with permanent hypoparathyroidism.
3
Laboratory tests showed lymphopenia with high LDH and hypocalcemia right after the birth.
4
Subsequent laboratory work demonstrated hypocalcemia, vitamin D deficiency, and secondary hyperparathyroidism.
5
The increment in serum iPTH produced by the EDTA-induced hypocalcemia was also unchanged.
6
Four asymptomatic episodes of mild hypocalcemia occurred in two patients.
7
AA levels have been measured in parathyroid glands from rabbits subjected to hyper- and hypocalcemia.
8
An idiopathic hypoparathyroidism-induced cardiomyopathy patient had severe long-lasting hypocalcemia.
9
His hypocalcemia was accentuated by the presence of hypomagnesaemia.
10
Objective: To provide family physicians with an evidence-based approach to the diagnosis and management of hypocalcemia.
11
And the risk factors associated with severe hypocalcemia (SH) in each group were analyzed.
12
Conclusion: Family physicians play a crucial role in educating patients about the long-term management and complications of hypocalcemia.
13
Perturbations can result in hypocalcemia or hypercalcemia and adaptations in calcium handling must occur during growth and aging.
14
We evaluated the biological potency and the availability for clinical use of this compound in hypocalcemia associated with hypoparathyroidism.
15
Another patient had a partial response for 5+ months, but developed symptomatic hypocalcemia, requiring discontinuation of the drug.
16
According to our analysis, both temporary and permanent hypocalcemia, which occurs in the postoperative period, are associated with incidental parathyroidectomy.