WKY rats decrease fatty acid synthesis and lipogenic processes whereas GK rats increase lipid elimination.

Acetate is the major carbon source spared and it may increase lipid synthesis in adipose tissue during MFD.

GIP acts on adipocytes to increase lipid uptake and enhance adipokine secretion, promoting weight gain and insulin resistance.

Gene expression profiling was consistent with increased lipid metabolism in adipose tissue.

Lipopolysaccharide increased lipid peroxidation most dramatically when it is injected 6 h before killing.

Both MRI and histology showed increased lipid composition and decreased fibrotic composition in these mice.

Increased lipid peroxidation may likely be a result of decreased endogenous antioxidant enzyme activities in TD.

In conclusion, ANGPTL4 deficiency increases lipid uptake and respiration in macrophages without affecting LPL protein levels.

This dosage also increased lipid oxidation in subcutaneous adipose tissue, thus causing a higher total energy expenditure.

The results tend to link central-central bridging fibrosis with increased lipid peroxidation and aldehyde-protein adduct formation caused by ethanol.

In addition, GF mice conventionalized with high-fat diet-induced jejunal microbiota exhibit increased lipid absorption even when fed a low-fat diet.

Here, we found that global genetic ablation of EHD2 in mice leads to increased lipid droplet size in fat tissue.

Conclusions: Our results suggest increased lipid turnover in the acute early phase of schizophrenia that is less obvious in chronic stages.

Antioxidant defense system alterations and increased lipid peroxidation have been postulated as a possible mechanism for neuronal damage associated with TD.

Plaques with increased lipid content appear more prone to rupture, particularly when the lipid pool is localized eccentrically within the intima.

Mechanistically, we elucidated that a combined activation of neutral and lysosomal (acid) lipolysis is responsible for the increased lipid degradation.