In WT mice, smoke exposure markedly increased pulmonary GM-CSF and AM accumulation.

Coinfection with MRSA and P aeruginosa is likely associated with further increased pulmonary morbidity.

Infected mice demonstrated increased pulmonary inflammation and alveolar epithelial cell apoptosis compared to controls.

Congenital heart diseases associated with increased pulmonary blood flow commonly leads to the development of pulmonary hypertension.

Inhaled nitric oxide may improve oxygenation by two mechanisms: increased pulmonary blood flow and improved ventilation-perfusion matching.

In contrast, at 48 h post infection, increased pulmonary leakage was apparent and not reversed by late antibiotic treatment.

We treated two patients with congenital heart disease and asthma who had increased pulmonary arterial pressure at cardiac catheterization.

Limited laboratory and clinical studies suggest a correlation between decreased plasma fibronectin levels and increased pulmonary capillary permeability and tissue perfusion.

Hemorrhaged mice resuscitated with MPs demonstrated substantially increased pulmonary neutrophil accumulation and altered lung histology compared with mice resuscitated without MPs.

The development of pulmonary hypertension is a common accompaniment of congenital heart disease (CHD) with increased pulmonary blood flow.

Shear stress secondary to increased pulmonary blood flow (PBF) is elevated in some children born with congenital cardiac abnormalities.

Surgeons approaching paraesophageal hernia repair should be aware of the increased pulmonary morbidity and the postoperative care required in managing these patients.

Objective: To investigate a possible link between increased pulmonary oxidative stress and the pivotal features of asthma during the mounting of an allergic inflammatory response.

Background: Right ventricular failure from increased pulmonary vascular loading is a major cause of morbidity and mortality, yet its modulation by disease remains poorly understood.

Thus, we conclude that L-carnitine may have a therapeutic potential in the treatment of pulmonary hypertension in children with CHD with increased pulmonary blood flow.

Increased pulmonary 67gallium uptake in scleroderma may prove useful as an index of pulmonary disease activity.