Значения термина induce nephrotoxicity на английском

A consensus set of phenotypic criteria for aminoglycoside- induced nephrotoxicity have recently been published.

Oxidative stress, inflammation and apoptosis are the important mechanisms of cisplatin induced nephrotoxicity.

Oxidative stress is the main mechanism responsible for cisplatin- induced nephrotoxicity.

This is the first study demonstrating the protective effect of gelofusine against colistin- induced nephrotoxicity.

The present work was undertaken to elucidate the pathophysiological mechanisms involved in cisplatin induced nephrotoxicity.

We suggest that the coadministration of OM might be beneficial for reducing of AA- induced nephrotoxicity.

The temporal relationship was strongly suggestive of etidronate- induced nephrotoxicity.

PC may have a protective effect against cisplatin- induced nephrotoxicity in rats via enhancing antioxidant enzyme activity.

This study sought to establish a sensitive and specific Miox-NanoLuc transgenic mouse for early detection of drug- induced nephrotoxicity.

Our findings suggest selective inhibition of AT1 receptors within the nephron as a promising intervention for protecting patients from cisplatin- induced nephrotoxicity.

The aim of this study was to characterize cisplatin- induced nephrotoxicity, oxidative stress in peripheral blood mononuclear cells, and the relationship between them.

This model may serve as an early detection for drug- and food- induced nephrotoxicity and as an animal model to investigate tubular cell injury.

Low-dose ADV- induced nephrotoxicity in most cases was reported to be reversible after the discontinuation of ADV or by decreasing the dose of ADV.

For vancomycin therapy of severe infections, the Infectious Diseases Society of America recommends high vancomycin trough levels, whose potential for inducing nephrotoxicity is controversial.

These cases illustrated that the use of low-dose ADV increased the risk of nephrotoxicity, and in some patients, low-dose ADV- induced nephrotoxicity was not completely reversible.

In conclusion, we developed a new platform to early quantify drug- induced nephrotoxicity before serum BUN and creatinine levels increased and pathological tubular cell injury occurred.