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Summary: Understanding the relationship between nutrient absorption and intestinal inflammation is important.
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Supplementation of IL-18 to Prevotella-colonized mice was sufficient to reduce intestinal inflammation.
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Paneth cell disturbances may instigate intestinal inflammation and provide susceptibility to infection.
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Conclusions: We found that tuft cell expansion reduced chronic intestinal inflammation in mice.
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Conclusions: The inhalation of CO protected mice from developing intestinal inflammation.
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Conclusions: F. saccharivorans decreased in correlation to UC activity and suppresses intestinal inflammation.
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Treatment with IL-10 resulted in a marked improvement in intestinal inflammation.
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HMPL-004-treated mice did not lose weight and displayed only very mild intestinal inflammation.
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IL-10-deficient mice spontaneously develop intestinal inflammation, which has many similarities to Crohn's disease.
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Chronic intestinal inflammation predisposes individuals to the development of colorectal cancer.
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Background: Probiotic bacteria have a beneficial effect on intestinal inflammation.
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These studies put βGalT1 forward as an essential protective player in exacerbated intestinal inflammation.
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Gelatinases may be useful targets for treatment of intestinal inflammation.
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Stool lactoferrin was used as a marker for intestinal inflammation.
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These T cell subsets might be potential biomarkers and therapeutic targets during intestinal inflammation.
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Metallothioneins have been linked to IBDs, but their role in intestinal inflammation is inconclusive.
