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On the other hand, the antagonists showed no significant effects on airway wall edema by LTC4.
2
A different pattern of symptoms was observed after in vivo nasal stimulation with histamine and LTC4.
3
The ratio of bronchial smooth muscle constriction by histamine was enhanced by the administration of LTC4.
4
The inhibition of human LTC4S will make a simple way to treat the cys-LT relevant inflammatory diseases.
5
Administration of LTC4 induced airway wall edema.
6
Liberation of histamine and LTC4 by cord-blood basophils was measured in the newborn from healthy and atopic parents.
7
These results suggest TXA2 may play a role in LTC4-induced bronchial hyperresponsiveness to histamine.
8
The whole-cell assay showed that compound 1 was cell permeable and inhibited LTC4 synthesis in a concentration dependent manner.
9
The LTC4 generation was well correlated with the appearance of allergic symptoms; LTB4 production was found in 2 patients only.
10
Additionally, the different expression patterns between molecules related to LTB4 and LTC4 synthesis suggested distinct roles of LTB4 and LTC4 in lung development.
11
We conclude that the immediate postnatal increases in TXB2 and PGI2 and decrease in LTC4 are not altered substantially by use of HFOV.