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Deleting p75 from the urothelium prevented urothelial death, but resulted in reduction in overall voiding efficiency after SCI.
2
The viral integrase protein interacts with transcriptional co-activator lens epithelium-derived growth factor p75 to principally position integration within gene bodies.
3
The neurotrophin receptor p75 is induced by various injuries to the nervous system, but its role after injury has remained unclear.
4
We further demonstrate that proNGF binds p75 in vivo and that disruption of this binding results in complete rescue of injured adult corticospinal neurons.
5
Here, we report that p75 is required for the death of oligodendrocytes following spinal cord injury, and its action is mediated mainly by proNGF.
6
Urothelial cell loss that precedes hyperplasia was dependent on p75 in response to urinary proNGF that is detected after SCI in rodents and humans.
7
The winner in each case taking P75, and the runner up P30, the remainder being divided amongst the most forward runners in the respective stakes.