Local passive immunity is the most efficient protective strategy to control the disease.

Maternal passive immunity regulated allergic airway inflammation in an FcRn-dependent manner.

Illness or vaccination during pregnancy confers passive immunity to the newborn.

In an enzootically infected colony, however, they are protected by maternally derived passive immunity.

Oral delivery of specific immunoglobulins provides passive immunity and is a fast acting treatment for rotavirus diarrhea.

The transportation of maternal antigen-specific IgG via amniotic fluid, placenta and breast milk plays an important role in passive immunity.

In addition, the gene expression patterns affected by the passive immunity transfer showed indicators that could benefit animal performance long term.

Maternal antibodies, specifically immunoglobulin G (IgG), are passed to the fetus to provide passive immunity throughout much of pregnancy.

Administration of colostrum to the newborn calf before gut closure is pivotal to its health, because of the transfer of passive immunity.

Potential roles for this MHC class I-like Fc receptor in the human intestine include the transfer of passive immunity, induction of oral tolerance, and immunosurveillance.