Значения термина phosphatidylserine asymmetry на английском

The breakdown of phosphatidylserine asymmetry is evidenced by annexin binding, a typical feature of apoptotic cells.

Consequences include breakdown of phosphatidylserine asymmetry, which depends at least partially on cellular loss of K+.

In nucleated cells cellular taurine is released prior to DNA fragmentation and the breakdown of phosphatidylserine asymmetry within the plasma membrane.

Similar to what is seen in nucleated cells, phosphatidylserine asymmetry is also abolished in erythrocytes exposed to osmotic shock or oxidative stress.

Erythropoietin binds to erythrocytes and inhibits volume-sensitive erythrocyte cation channels and thus the breakdown of phosphatidylserine asymmetry after activation of this channel.

Erythrocyte cell volume was estimated from forward scatter and breakdown of phosphatidylserine asymmetry was identified by determination of annexin binding using FACS analysis.