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1
The majority of pathogenic mutations in BRCA1 result in a
truncated
protein
.
2
By contrast, this helix adopts a perpendicular orientation in the
truncated
protein
.
3
Although most lead to a
truncating
protein
,
the severity of the phenotype differs.
4
None of the
truncated
proteins
retained full transactivating ability on the RANTES promoter.
5
The mutation is predicted to encode a
truncated
protein
or result in nonsense-mediated decay.
6
The latter is expected to escape nonsense-mediated mRNA decay resulting in a
truncated
protein
.
7
Truncated
proteins
encoded by these DI RNAs were indeed detected in the nucleocapsid preparations.
8
Four new transcripts that code for
truncated
proteins
lacking the collagenous domain were also detected.
9
These mutations will lead to production of
truncated
protein
.
10
The short transcript partially escaped nonsense-mediated mRNA decay and was translated into a
truncated
protein
.
11
The mutation results in a
truncated
protein
of PRPF31.
12
The identified deletion resulted in a frame shift producing a
truncated
protein
of 208 aa.
13
The shed CD31 can be specifically detected as a soluble,
truncated
protein
in human plasma.
14
These mutations lead directly or indirectly to premature stop codons, and the mutations generate
truncated
proteins
.
15
Some mutants specified a
truncated
protein
,
whereas others completely lost the ability to specify the protein.
16
Most crystal forms of the
truncated
proteins
were isomorphous with those of the virally extracted protein.